By F. Z. Meerson (auth.), E. Chazov M.D., V. Saks M.D., G. Rona M.D. (eds.)
This quantity of Advances in Myocardiology is derived from part of the lawsuits of the tenth Congress of the overseas Society for center examine, which was once held in Moscow on September 23-29, 1980. This e-book comprises chosen papers that have been prepared in sections, Cardiac Hypertrophy, edition, and Pathophysiology and Cardiac Hypoxia, Is chemia, and Infarction. the 1st part, at the pathophysiology of middle hypertrophy and failure, includes 24 chapters that concentrate on the derangement of biochemical, physiological, and immunological procedures through the de velopment of center sickness as a result of a wide selection of pathogenic elements. a few of the fresh advancements in realizing the myocardial man made ma chinery in middle hypertrophy also are defined in those papers, and we think that the contents of this part will stimulate extra learn within the quarter of middle disorder. the second one part, which typically offers with myocardial ischemia, includes 35 chapters supplying the mandatory again flooring for the analysis of ischemic middle affliction and a few attainable ther apeutic ways. It rarely has to be emphasised that ischemic center illness is an immense reason behind demise in hugely industrialized nations, yet regrettably the precise mechanism wherein ischemic insult results in the advance of middle mobilephone harm is way from understood. we're hopeful that the articles during this part will supply important info during this box and hence might help in bettering the remedy of ischemic center disease.
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Extra resources for Advances in Myocardiology: Volume 4
Mollg) • See Figure 2 for experimental design. , 10 min after addition of drug. Dashes indicate absence of data. b Data from Horak et al. (12). C Compared with value at 30 min of perfusion. d Values at 45 min in control hearts are taken as 100%. e Cumulative values at 75 min same as in control. See Figure I. f See discussion in text. I. Control value 2. Control % change at 35 min 3. Epinephrine 10- 8 M 4. Epinephrine 10- 7 M 4. Epinephrine 10- 6 M 6. Theophylline 10- 3 M 7. Dibutyryl cAMP 10- 4 M 8.
II: 1073-1094. Reuter, H. 1974. Localization of l3-adrenergic receptors, and effects of noradrenaline and cyclic nucleotides on action potentials, ionic currents and tension in mammalian cardiac muscle. J. Physiol. ) 242:429-451. , Chappel, C. , and Gaundry, R. 1959. An infarct-like myocardial Metabolism in Catecholamine Injury 26. 27. 28. 29. 30. 31. 32. 33. 43 lesion and other toxic manifestations produced by isoproterenol in the rat. Arch. Pathol. (Chicago) 67:443-445. Sutherland, E. , Robison, G.
Opie, L. , and Bricknell, O. L. 1979. Role of glycolytic flux in effect of glucose in decreasing fatty-acid-induced release of lactate dehydrogenase from isolated coronary-ligated rat heart. Cardiovasc. Res. 13:693-702. Opie, L. , Mansford, K. R. , and Owen, P. 1971. Effects of increased heart work on glycolysis and adenine nucleotides in the perfused heart of normal and diabetic rats. Biochem. J. 124:475-490. Opie, L. , and Owen, P. 1975. Assessment of mitochondrial free NAD+ INADH ratios and oxaloacetate concentrations during increased mechanical work in isolated perfused rat heart during production or uptake of ketone bodies.
Advances in Myocardiology: Volume 4 by F. Z. Meerson (auth.), E. Chazov M.D., V. Saks M.D., G. Rona M.D. (eds.)